Thomson, William Murray van der Putten, Gert-Jan de Baat, Cees Ikebe, Kazunori Matsuda, Ken-ichi Enoki, Kaori Hopcraft, Matthew Ling, Guo Y In all stages, they should keep treatment noninvasive if possible. As xerostomia progresses, they should shift focus primarily to prevention, maintenance, oral comfort, and emergency treatment. Including a dentist on the treatment team is essential for residents with dry mouth.
Cancer, autoimmune diseases, and bone marrow transplants are associated with xerostomia. Medications-several hundred of them-can cause or exacerbate xerostomia. Xerostomia can lead to digestive problems, weight loss, and accelerated dental decay. Saliva is necessary for digestion and dental health, and it may have yet-unidentified immunological roles in humans. Up to 30% of various populations self-report dry mouth ( xerostomia) or have proven low salivary flow rates. Non-muscle symptoms tend to be moderate and are often diagnosed and treated as independent issues (e.g., cataracts or frontal balding in men).These individuals are unlikely to be diagnosed as having myotonic dystrophy unless a family member is identified with a more severe form of the disease and testing is initiated.When reduced salivary flow causes perpetual dry and sticky mucosa or sticky, stringy saliva, it becomes xerostomia-not a disease, but a symptom. Muscle symptoms (if any) may be attributed to general stiffness or arthritis. Depending on severity of symptoms, patients with adult DM1 may be categorized as having either the mild or classical form of the disease. Individuals with the mild form of adult-onset DM1 are often not aware they have the disorder. With adult-onset DM1, symptoms can appear from late adolescence through old age, and usually worsen over time.
Adult Onset: Characterized by distal muscle weakness, atrophy, myotonia and many other multisystemic issues.The time of onset is often dependent on the astuteness of the observer, and is typically first presents with intellectual disability, and learning disabilities. After a relatively normal birth and infancy, individuals with childhood-onset DM1 develop symptoms sometime between early childhood and early adolescence. Childhood Onset: Typically first presents with intellectual disability, and learning disabilities.
(In DM2, there have been no reported cases of a congenital form). During pregnancy, she may have noticed less than normal levels of fetal activity, had a condition called hydramnios where excess amniotic fluid (edema) accumulates, or had a long, difficult labor. It occurs when a mother, who often is not aware she has DM1, passes the mutation that causes DM1 onto her child.
0 kommentar(er)
